Preliminary scientific tests indicate conolidine may possibly inhibit unique ion channels, lessening neuronal excitability and restricting soreness alerts. This mechanism is particularly suitable in neuropathic ache, wherever irregular signaling leads to persistent pain. Furthermore, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to suffering percepti... https://catmanx951sja7.wikistatement.com/user