Preliminary studies indicate conolidine could inhibit specific ion channels, decreasing neuronal excitability and restricting soreness signals. This mechanism is especially suitable in neuropathic suffering, where irregular signaling causes persistent pain. In addition, conolidine seems to impact G protein-coupled receptor (GPCR) pathways integral to ache notion. Computational docking... https://australianm958vhh7.p2blogs.com/profile