Metabolic alterations affiliated with T2D are very well characterised by epidemiological and research centered research. The pathogenesis and progression of T2D is ascribed to 4 mechanisms; increased Innovative glycation end merchandise (AGE) formation, enhanced polyol pathway flux, activation of protein kinase C (PKC) isoforms, and greater hexosamine pathway flux[seven]. Till https://www.directivepublications.org/journal-of-diabetology-research/